| Application | Loss weight |
| Size | 2mg,5mg,10mg,15mg |
| MOQ | 1 kit(1 kit is 10 vials) |
| Purity | >99% |
| CAS | 910463-68-2 |
| Shipping time | 15-20 days |
| Payment | Western unition ,bank transfer,BTC,ETH, Paypal |
99.9% purity semaglutide 2mg 5mg 10mg 15mg lose weigth peptide GLP-1
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Semaglutide is the active ingredient in several diabetes and weight loss drugs lThis compound is a synthetic form of the naturally occurring hormone called glucagon-like peptide-1 (GLP-1).
Developed by Novo Nordisk, semaglutide was initially approved by the U.S. Food and Drug Administration (FDA) in 2017 as a part of the approval process for Ozempic, an injectable medication used to treat type 2 diabetes in adults.
In 2019, a new form of semaglutide was FDA-approved under the brand name Rybelsus. Rybelsus was the first-ever oral GLP-1 receptor agonist in American markets for treating type 2 diabetes.
Most recently, in 2021, the FDA approved a more potent dose of injectable semaglutide, this time under the brand name Wegovy. Unlike the other two forms of semaglutide, which cause weight loss but aren’t explicitly FDA-approved weight loss aids, Wegovy is specifically indicated to help individuals with obesity or overweight individuals with weight-related health problems lose weight. It is prescribed in conjunction with a healthy diet and regular exercise.
Because of rising type 2 diabetes and obesity rates around the globe, semaglutide use is skyrocketing. Semaglutide prescriptions have gone up fortyfold in the last five years. About 1.6% of the US population takes a form of semaglutide medication. However, according to some studies, over 53% of the US population would be considered eligible for a semaglutide prescription.
Semaglutide’s multiple benefits can help lower blood sugar, reduce body weight, and lower the risk of cardiovascular events—like heart attack, stroke, or heart disease—in millions of patients.
Semaglutide is a synthetic compound that imitates the effect of the natural hormone GLP-1 in the body. Semaglutide is officially classified as a glucagon-like peptide-1 receptor agonist, and its impact on blood sugar metabolism and calorie intake is multiple.
After eating a meal, your intestines release a hormone called GLP-1, which creas to release more insulin. Insulin is a hormone that plays a crucial role in reducing blood sugar levels by facilitating the entry of glucose (sugar) into , where it can be used for energy or stored for later use. GLP-1 is especially important in ensuring that insulin is released at the right times, particularly after meals when blood sugar levels typically rise.
In addition to its role in insulin regulation, GLP-1 also has the added benefit of reducing the secretion of another hormone, glucagon, produced by the pancreas. Unlike insulin, glucagon has the opposite effect, increasing blood sugar levels by instructing the liver to release stored glucose into the bloodstream. By inhibiting glucagon, GLP-1 prevents the excessive release of glucose from the liver, thus keeping blood sugar levels from becoming excessively high.
GLP-1 also has an impact on the rate at which your stomach empties. When the pancreas releases GLP-1, it helps control the pace at which food moves from the stomach to the small intestine. This action can slow down the absorption of nutrients, including glucose (sugar), from the digestive tract into the bloodstream. Consequently, this prevents rapid spikes in blood sugar levels after meals. Additionally, slower stomach emptying can create a feeling of fullness, reducing overall food intake and contributing to weight management.
It should be noted that higher doses of semaglutide are more likely to cause significant weight loss than the lower doses that are administered via Rybelsus or Ozempic. In addition, semaglutide is most effective as a chronic weight management aid when combined with lifestyle changes like a reduced-calorie diet and regular exercise.
When taken correctly, semaglutide can affect weight loss through two primary actions: blood sugar management and appetite regulation.
Semaglutide stimulates the pancreas to release insulin. This hormone is crucial in transporting glucose (sugar) from the bloodstream into , where it can be utilized for energy. By facilitating this process, semaglutide helps maintain stable blood sugar levels, reducing the likelihood of sudden energy fluctuations that can trigger cravings for high-calorie foods.
Furthermore, semaglutide effectively inhibits the production of glucagon, another hormone responsible for elevating blood sugar levels. Glucagon prompts the liver to release stored glucose into the bloodstream, but semaglutide intervenes in this process. By controlling glucagon, semaglutide helps keep blood sugar levels steady, mitigating the hunger pangs often caused by low blood sugar.
Semaglutide also influences appetite by interacting with GLP-1 receptors in the brain, particularly in the hypothalamus. When semaglutide binds to these receptors, it n to reduce appetite and curb cravings. This means that individuals taking semaglutide tend to experience reduced hunger and greater satisfaction from smaller meals.
Additionally, semaglutide slows down the rate at which the stomach empties its contents into the small intestine. This delay in stomach emptying leads to prolonged feelings of fullness after eating, ultimately reducing the inclination to consume more food.
It's important to note that semaglutide is typically prescribed with lifestyle modifications such as calorie reduction and increased sical activity. It is not intended to be the sole treatment for obesity but rather a supportive component of a comprehensive approach to weight management.
From October 5, 2018, to February 1, 2019, a study was conducted with 304 participants. They were randomly assigned to receive either semaglutide (152 participants) or a placebo (152 participants). This study looked at their body weight and various health parameters. Most of the participants were female (77.6%) and white (93.1%), with an average age of 47.3 years, an average weight of 106.0 kg, and an average body mass index—BMI—of 38.5 kg/m².
The study used two different approaches to analyze the effectiveness of semaglutide. The first approach, the 'treatment policy' estimand, looked at how semaglutide worked for all participants throughout the study, regardless of whether they stopped taking the medication or received other weight loss treatments. The second approach, the 'trial product' estimand, focused on how semaglutide worked when participants took it as intended.
Based on the 'treatment policy' estimand, semaglutide significantly reduced body weight compared to the placebo after 104 weeks. Participants taking semaglutide lost an average of 15.2% of their initial body weight, while those on the placebo only lost 2.6%. Semaglutide users were also more likely to achieve a weight loss of at least 5%. The study concluded that semaglutide was effective in helping participants lose weight.
Semaglutide also positively affected waist circumference, blood pressure, blood sugar levels, and other health markers. For example, it improved both systolic and diastolic blood pressure. It also positively affects glycated hemoglobin, fasting plasma glucose, and lipid profiles. These effects can reduce the risk of cardiovascular disease or events like heart attack or stroke.
In participants with prediabetes at the beginning of the study, semaglutide helped a significant of them revert to normal blood sugar levels. Conversely, in participants with normal blood sugar at the start, very few developed prediabetes. None of the participants on semaglutide developed type 2 diabetes during the study.
In summary, the study found that semaglutide was effective in helping participants lose weight and improving various health parameters, making it a promising treatment for obesity and related health conditions.
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